Living with Crohn's Disease

Abstract

The study discusses Crohn's disease and important implications that patients need to consider. Living with Crohn's disease requires patients to understand the complications, causes and its treatment. Furthermore, the research also discusses the the environmental factors that may complicate conditions for patients living with Crohn's disease.

Table of Contents

Abstractii

Introduction1

Discussion1

Treatments2

Perspectives3

Symptoms of the disease5

Causes of Crohn's disease5

Complications of Crohn's disease6

Medical examinations7

Tips for Crohn's disease patients7

Food for patients with Crohn's disease7

Amenities8

What are the possible mechanisms of Crohn's disease?9

A genetic approach to Crohn's disease9

Environmental factors in the disease10

Psychological factors:11

Oral contraceptives:11

The anti-inflammatory drugs:11

Factors that triggering Crohn's disease:11

Role of specific infectious agents:11

Role of infectious agents or non-specific Flora of the digestive tract:12

Conclusion13

References14

Living with Crohn's Disease

Introduction

Crohn's disease is a chronic disease of unknown origin that may have a component autoimmune in which, the immune system attacks its own individual's intestine causing inflammation. In most cases, the affected part is the ileum or final leg of the small intestine, although the disease can occur anywhere in the digestive tract. The disease was first described by Giovanni Battista Morgagni (1682-1771). John Berg, in 1898, and the Polish surgeon Antoni Lesniowski, in 1903, described more cases of the disease. In 1932, the information was published again for Burrill Bernard Crohn and his colleagues Ginzburg and Oppenheimer, and the first surname comes from the biggest name of the disease. Sometimes, it can be found under the name of regional enteritis or granulomatous colitis. The exact origin of the disease is unknown, but is known about genetic and factors that increase environmental risk. Several studies have linked the disease to genetic variants, including a gene on chromosome 16, but it is not strictly hereditary.

Discussion

This evil belongs to the group of inflammatory bowel disease (IBD, IBD), which is also part of ulcerative colitis. Since mid 2006, the call is accepted classification of Montreal, which was proposed by the World Organization of Gastroenterology, which classifies patients according to age at onset, disease location and behavior of this. As for the age at onset, types are distinguished A1 (under 16), A2 (16 to 40) and A3 (over 40). In terms of location, L1 (ileum and perhaps neighboring areas of blind), L2 (colon only) and L3 (sum of L1 and L2), also added "+ L4" if the patient has affected the upper gastrointestinal tract ( esophagus , stomach , duodenum , jejunum or proximal ileum). In behavior, we define B1 (type inflammatory predominate manifestations of inflammation), B2 (type stenotic, predominantly stenosis or strictures in the gastrointestinal tract) and B3 (penetrating type, dominated by fistulas and fissures). In any case, if there is involvement of the anal region, add "+ P" (Sartor, 1995). Thus, a person with the disease starting at age 30, has affected the colon and esophagus, and has a pattern of bowel strictures and also fistulas in the year , has pattern A2, L2 + L4, B2 + p .

Treatments

Photopheresis treatment: the patient is subjected to photopheresis procedure whereby positive trend has been observed in a significant number of ...