PRINCIPLES OF ENDODONTICS

Principles of Endodontics

Root Canal Resorption

Introduction

In the field of dentistry, root resorption is referred to the destruction or breakdown, and the subsequent loss of the root structure of a tooth. This destruction is caused by the living body cells hat attack the part of a tooth. It is very important to understand that when the damage extends to the whole tooth, then it is known as tooth resorption. It is also obvious that severe form of root resorption is very difficult to treat and could also need the extraction of teeth. Root resorption is basically a result of differentiation of macrophages into odnotoblasts in surrounding tissue which in close proximity to the rot surface will aim to resorb the root surface cementum and the underlying root dentine. This could vary in severity from the evidence of microscopic pits in the root surface for reaching the complete devastation of the root surface. The core focus of this paper is to discuss a classification of the root canal resorption. The treatment of one of the selected classification of root canal resorption and the outcome of the treatment would also be discussed in the following discussion.

Discussion

The classification that has been selected for analysis in this paper is inflammatory root resorption along with a thorough discussion and it needs a great focus for effective treatment. The following is an account of the basic definition along with the treatment and outcomes of inflammatory root resorption.

Inflammatory Root Resorption

Inflammatory root resorption is a common complication occurring after the offset tooth, for example as a result of subluxation or complete dislocation. Extrusion or intrusion of the tooth, and subsequent manipulations aimed at its reposition inevitably lead to damage to the tissue surrounding the root, resulting in the surface portions formed dentin nude providing chemotaxic effect on phagocytes. It may start transient root resorption. Furthermore, the shift of the teeth can lead to rupture of blood vessels in the pulp area of the apical foramen to ischemic necrosis of the pulp (Walton, 2009). Thus through the enamel-cement cracks and exposed dentine is penetration of microorganisms in the root canal, resulting in a few days there is inflammation. In this transient root resorption that occurs in response to stimulation by the damaged sections of the root, leading to exposure of root dentin. Dentine tubules on the waste products of bacterial cells come from the infected root canal defect region, thus supporting the resorption of the root.

In this regard, the trigger of the outer root resorption is a mechanical injury, leading to destruction cementoblast, pretsementa and sometimes cement on the root surface. In the future, the process of resorption is maintained by constant irritation resorbing cells of bacterial products coming from infected root canals. Radiographic changes in the form of X-ray illumination juxtahilar hearth, exciting tooth root and the adjacent alveolar bone, there a few weeks later (see Fig. 8.16). If the development process is not suspended, progressive resorption can last several months lead to the complete ...